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These along with telomere damage, as a result of oxidative stress, lead to upregulation of the cell cycle inhibitor p16 INK4 and mammalian target of rapamycin (mTOR)-mediated signalling to trigger premature senescence in COPD airway epithelial cells (145149)

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The mitophagy receptor BNIP3 also functions as a tumor suppressor in mammary and pancreatic tumorigenesis [237, 238], illustrating mitophagys crucial role in cancer regulation

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