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During pulldown experiments to identify TG2-interacting proteins and to analyze domaindomain interactions, lysates from cells that express a high level of TG2 exhibited an increase in aggregate formation

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GLP-1 RAs in Steatotic Liver Disease (MASLD, MetALD, and ALD) Although not yet specifically approved for the treatment of MASLD, GLP-1 RAs exhibit pleiotropic effects that contribute to the improvement of MASLD and its progression to metabolic dysfunction-associated steatohepatitis (MASH) (Table 3)